brain disease
Inflammatory response of brain cells -- as indicated by a molecular imaging technique -- could tell researchers more about why certain neurologic disorders, such as migraine headaches and psychosis in
(BOSTON) -- The Center for the Study of Traumatic Encephalopathy (CSTE) at Boston University School of Medicine (BUSM) announced today that a recently deceased member of the NFL Hall of Fame suffe
(BOSTON) -- The Center for the Study of Traumatic Encephalopathy (CSTE) at Boston University School of Medicine (BUSM) announced today that a deceased former college football player who died at age 42 was already suffering from the degenerative brain disease, Chronic Traumatic Encephalopathy (CTE).
BOSTON -- The virus responsible for PML (progressive multifocal leukoencephalopathy), a rare brain disease that typically affects AIDS patients and other individuals with compromised immune systems, has been found to be reactivated in multiple-sclerosis patients being treated with natalizumab (Tysabri).
A new study finds substantial improvement in a mouse model of a rare, hereditary neurodegenerative disease after transplantation of normal human neural stem cells.
July 22, 2009 -- (BRONX, NY) -- A cellular protein that may prevent nerve cells from dying also helps to improve insulin action and lower blood glucose levels, according to a study by researchers at Albert Einstein College of Medicine of Yeshiva University (http://www.aecom.yu.edu) in collaboration with scientists at Unive
DURHAM, N.C. - Scientists at Duke University and the University of North Carolina have devised a chemical technique that promises to allow neuroscientists to discover the function of any population of neurons in an animal brain, and provide clues to treating and preventing brain disease.
ST. LOUIS -- In a multi-center trial led by a Saint Louis University researcher, investigators found that a new combination therapy of daily consensus interferon and ribavirin helps some hepatitis C patients who have not responded to previous treatment.
LA JOLLA, CA, June 8, 2009 ?As survival rates among some patients with cancer continue to rise, so does the spread of these cancers to the brain -- as much as 40 percent of all diagnosed brain cancers are considered metastatic, having spread from a primary cancer elsewhere in the body.
GAINESVILLE, Fla. -- University of Florida scientists have discovered why a paralyzing brain disorder speeds along more rapidly in some patients than others -- a finding that may finally give researchers an entry point toward an effective treatment for amyotrophic lateral sclerosis, often referred to as ALS or Lou Gehrig's disease.
CHICAGO --- The 57-year-old lawyer in New York had handily completed the New York Times' Saturday crossword puzzle ? the hardest of the week ? for years. But one Saturday morning, suddenly he couldn't retrieve the words to fill in the squares.
Of dozens of candidates potentially involved in increasing a person's risk for the most common type of Alzheimer's disease that affects more than 5 million Americans over the age of 65, one gene that keeps grabbing Johns Hopkins researchers' attention makes a protein called neuroglobin.
Researchers at NYU School of Medicine have found that immunization prolongs the incubation period for prion diseases such as Creutzfeldt-Jakob disease and may have therapeutic value for other neurodegenerative illness such as Alzheimer's disease. Prion disease is a fatal brain disease manifested through failure of muscle control and dementia. Forms of this disease have been discovered in deer and elk (chronic wasting disease), in cows (bovine spongiform encephalopathy ? BSE ? or "mad cow disease") and in sheep (scrapie strain).
Greater insight into human brain disease may emerge from studies of a new genetic mutation that causes adult fruit flies to develop symptoms akin to Alzheimer's disease. "This is the first fruit fly mutant to show some of the outward, physical manifestations common to certain major human neurodegenerative diseases," said principal investigator Michael McKeown, a biology professor at Brown University. A research team found the mutation in a gene they named "blue cheese."
Agents that alter blood levels of beta-amyloid protein in mouse models of Alzheimer's disease represent a potential approach to treating the illness in humans that may be safer than the vaccine method of therapy, researchers report in a new study. Beta-amyloid protein is a component of the amyloid plaques that accumulate in the brains of people with Alzheimer?'s disease. Beta-amyloid is viewed by many researchers and clinicians as the underlying cause of the degeneration and dementia that characterize the illness. Alzheimer's disease is a progressive, degenerative brain disease and the most common form of dementia. There is no cure.
